Article: The Muscoloskeletal Effects of Diabetes Mellitus [Journal of the Canadian Chiropractic Association]

“The purpose of this paper is two-fold. Chiropractors see patients with both types of DM [Diabetes Mellitus]. It is important for the practicing chiropractic doctor to recognize the effects of DM on the musculoskeletal system so as to make more appropriate clinical decisions regarding therapy in these patients, including understanding contraindications to therapy and referring patients to medical physicians when appropriate. It is also important for the DC [Doctor of Chiropractic] to understand the impact that DM may have on the prognosis for their patients suffering from myriad musculoskeletal conditions associated with this disease.

“In addition, it has been suggested that health care providers offer their patients counseling to promote physical activity, a healthy diet, and smoking cessation as part of the preventive health examination. It is also in this light that the musculoskeletal effects of DM are discussed in this manuscript, as most patients visiting chiropractors have musculoskeletal complaints. , It is hoped that through appropriate counseling regarding a healthy lifestyle, the chiropractor may help to prevent some cases of Type 2 DM and that the incidence of the musculoskeletal effects of DM can be reduced.”

“This manuscript will discuss some of the more common musculoskeletal manifestations of diabetes mellitus. Table 2 lists the more common musculoskeletal effects of DM: muscle cramps, muscle infarction, loss of deep tendon reflexes, peripheral neuropathy, Reflex Sympathetic Dystrophy Syndrome, Stiff Hands Syndrome, neuropathic joints, Carpal Tunnel Syndrome, adhesive capsulitis of the shoulder, tenosynovitis, Diffuse Idiopathic Skeletal Hyperostosis, Dupuytren’s contracture.”

Read the full paper by Lawrence H. Wyatt and Randy J. Ferrance at NCBI: The Musculoskeletal Effects of Diabetes Mellitus

 

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Article: Chronic Migraine Worsened by TMJ Disorder [Fascia & Fitness]

Fascia & Fitness does a marvelous job of making the results of a study in Brazil easier to read (especially when you currently have a headache).

“The lead author Lidiane Florencio wrote regarding the link between TMJ disorder (“TMD”) and migraines: “The repetition of migraine attacks may increase sensitivity to pain. Our hypothesis is that migraine acts as a factor that predisposes patients to TMD. On the other hand, TMD can be considered a potential perpetuating factor for migraine because it acts as a constant nociceptive input that contributes to maintaining central sensitization and abnormal pain processes. Nociceptive pain is caused by a painful stimulus on special nerve endings called nociceptors.”

“Migraine and TMJ disorder have very similar pathologic mechanisms. Migraine affects 15% of the general population, and progression to the chronic form is expected in about 2.5% of migraine sufferers. On the other hand, TMJ disorder is stress-related as much as it has to do with muscle overload. Patients display joint symptoms – such as joint pain, reduced jaw movement, clicking or popping (crepitus) of the temporomandibular joint – but also develop a muscular condition, including muscle pain and fatigue, and/or radiating face and neck pain.

“TMJ disorder and migraine often co-occur. However, while people who suffer from migraine are more likely to have TMJ disorder, people with TMJ disorder will not necessarily have migraine. The researchers believe that TMJ disorder may increase the frequency and severity of migraine attacks, even though it does not necessarily directly precipitate the migraine attack.”

Read the full article by Fascia & Fitness: Chronic Migraine Worsened by TMJ Disorder

Read the published study in the Journal of Manipulative and Physiological Therapeutics: Association Between Severity of Tempomandibular Disorders and the Frequency of Headache Attacks in Women with Migraine: A Cross-Sectional Study

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Article: Where Does Somatic Memory in the Body Reside? [Fascia & Fitness]

“Traumatic memories are often experienced as “relived” rather than remembered, which is why people experiencing them react as though they are re-experiencing the situations in which they were traumatized. When a traumatic memory is triggered, the somatosensory experience of the person reliving the memory can be powerful; the whole body “remembers” and replicates the sensations of the trauma, including sympathetic nervous system fight, flight, or freeze responses. The psychophysiological experience is of reliving the trauma, what we call a flashback. In this situation, the client often effectively dissociates from the present reality and is caught in the state of re-living the traumatic memory.

“Whereas memories of ordinary events, even those containing somatosensory and emotional components, do not have the somatosensory texture and depth of flashbacks, making it much easier to remain connected to external stimuli and to experience being present in the moment while simultaneously feeling (remembered) sensations or emotions.”

[The article continues with remarks from Til Luchau, who I desperately want to train with some day. I have to be content with his Advance Trainings fb group for the time being.]

The state-dependant memory model discussed above [not included in this excerpt, read the full article] is more nuanced and sophisticated, and so arguably more useful. It brings to mind a book I’m currently reading: Lisa Feldman Barrett’s How Emotions are Made (2017, Houghton Mifflin Harcourt. ISBN 9780544133310). In her “theory of constructed emotions,” Barrett builds on the idea that our brains are structured to predict what we will see, taste, here, and feel. Apparently, there’s good evidence that the brain only processes things it does not predict. In this model, preloaded but widely networked caches of information (concepts) and meaning (valence) are used to minimize the brain’s energy use and maximize processing time.

“Interestingly, she writes that the brain’s wiring causes internal sensation and body signals (interoception and proprioception) to reach the brain’s processing centers before external perceptions (exteroception), such as sight, hearing etc. This sets up the brain to rapidly predict what it’ll perceive exteroceptively, based largely on past bodily experience (as well as language) what’s going to happen outside. In other words, we take in sensory information only until our brains can predict what will happen.
 
“This is the proposed mechanism behind both perceptions and emotion: for example, in this model, we are not reacting to our perceptions with emotions, we are neurologically predicting what will happen, and it is our predictions that shape our perceptions, emotions, and actions.”
 
Read the full article (and Til’s full commentary, plus comments from Walt Fritz) from Fascia & Fitness: Where Does Somatic Memory in the Body Reside?
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Article: Just the Two of Us: Holding Hands Can Ease Pain, Sync Brainwaves [CU Bolder Today]

“We have developed a lot of ways to communicate in the modern world and we have fewer physical interactions,” said lead author Pavel Goldstein, a postdoctoral pain researcher in the Cognitive and Affective Neuroscience Lab at CU Boulder. “This paper illustrates the power and importance of human touch.” 

“The study is the latest in a growing body of research exploring a phenomenon known as “interpersonal synchronization,” in which people physiologically mirror the people they are with. It is the first to look at brain wave synchronization in the context of pain, and offers new insight into the role brain-to-brain coupling may play in touch-induced analgesia, or healing touch.

“Goldstein came up with the experiment after, during the delivery of his daughter, he discovered that when he held his wife’s hand, it eased her pain.

“I wanted to test it out in the lab: Can one really decrease pain with touch, and if so, how?”

Read Lisa Marshall’s full article at CU Boulder Today: Just the Two of Us: Holding Hands Can Ease Pain, Sync Brainwaves

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